Babraham Institute Theses 2005

(University of Cambridge unless otherwise stated)

Anderson NF The role of the imprinted gene Peg1 in mammalian development.

Choudhury SK Neuropathological and molecular analyses of a transgenic mouse model over-expressing a C-terminal fragment of human Familial Alzheimer's disease APP.

Coar EA Mapping candidate control elements at the Gnas imprinted locus.

Cobb LJ Roles of insulin-like growth factor binding protein-5 (IGFBP-5) during myogenesis.

Cuddon P The role of Ca2+ influx in neuronal cell growth and function: a study of hippocampal neurons and neurosphere-derived cells.

Daly JN IgH allelic exclusion at the level of IgH transcription.

Delon C The identification and characterisation of novel phosphatidate binding proteins.

Densham RM The integration of growth and stress signalling at the G1[right arrow]S transition.

Hill K Regulation of P-Rex1 by PtdIns(3,4,5)P3 and Gβγ subunits.

Holmes AM The ins and outs of Ca2+ signalling: regulation of Ca2+ entry and release in electrically non-excitable cells.

Khan MR The role of Deltex1 in lymphocyte development.

Maqueira B Cloning and characterization of a novel family of β-adrenergic-like octopamine receptors from Drosophila melanogaster.

Maxwell GM A study of the mouse precursor B cell receptor (pre-BCR) signal.

Miles JN Intergenic transcription and chromatin remodelling of the human β-globulin locus.

Norgate Z Gene expression in rat uterine natural killer cells.

Parker MJD Control of growth and performance of precursor B lymphocytes.

Sanchez-Andrade G Ovarian cycle influences on neural plasticity and olfactory learning in the mouse.

Srivastava DP Characterisation of a novel Drosophila G-protein coupled receptor differentially activated by catecholamines and steroids.

Tkachev D Expression profiling in schizophrenia and bipolar affective disorder.

Wilson MF Investigating the role of cis-acting sequences in the imprinting of the human GNAS1 locus using a transgenic model.

Wood EJ Phospholipase D1 dynamics in animal cells.